DOI: 10.36347/sjams.2018.v06i06.024

Pages: 2432-2435

The pathways involved in brain-heart interaction have been explicated in both animal and human studies. Neurogenic mechanism suggested that, sympathetic nervous system stimulation can produce the arrhythmia by its activation. In addition both anatomical and physiological evidence implicated the hypothalamus in cardiac control. Several experimental studies reported that electrical stimulation of posteriorly located area of cardiovascular sympathetic control and anterior parasympathetic control region. Cardiac dysfunction after cerebrovascular accidents is manifested by ECG changes, cardiac arrhythmias, elevated cardiac biomarkers and hemodynamic alteration. Present study was taken to know the incidence and pattern of ECG changes in patient with cerebrovascular disease. Total 56 cases of cerebrovascular disease patients were studied to know the ECG changes. They were categorized into 3 different cerebrovascular diseases based on the CT scan findings as cerebral infarction, cerebral hemorrhage and subarachnoid hemorrhage. Out of 56 patients, 69.6% of all cerebrovascular disease patients had some form of ECG abnormalities. Higher incidences of abnormal ECG found in cerebral hemorrhage patients 72.7%. Sinus bradycardia was seen in 18.1% of cerebral hemorrhage patients, none of the patients had sinus bradycardia in cerebral infarction and subarachnoid hemorrhage. The study was concluded that ECG changes in cerebrovascular disease patients were not associated with any particular site of cerebral lesion but rhythm changes in pattern of ECG were specific as categorized by CT scan findings.